Answer:
A i think if not im sorry
Explanation:
Answer:
The correct answer is: a basement membrane.
Explanation:
The basement membrane is a thin and fibrous membrane that is found under the epithelium and<u> attaches it to the connective tissue</u>. The basement membrane is <u>composed of two membranes</u>: the basal lamina (with laminin and collagen IV, among other proteins), and the lamina reticularis (that has collagen III making up reticular fibers). The basement membrane also has the function of a <u>mechanical barrier that prevents the invasion of deeper tissues by malignant cells</u>.
Epithelial tissue and connective tissue are two of the four basic tissues found in the body (the other two are nervous tissue and muscle tissue). Epithelial tissue makes up the skin and the <u>inner lining of hollow organs</u> (like the stomach or the trachea), and has multiple functions that are responsible for the function of the organ where it's located. Connective tissue is basically any type of tissue that connects the epithelium to muscle tissue or nervous tissue: <u>from blood, to bone, and to fat</u>.
Answer:
The correct answer would be 93 milliliters.
The same can be explained with the help of the unitary method.
The volume of solution required for 1 kg of weight in one hour = 2.5 milliliters (given in the question).
Thus, the volume of the solution required for 37.3 kg of weight in one hour = 
milliliters
It comes out be 93.25 milliliters.
Thus, around 93 milliliters of the solution should be given to the child of weight 37.3 kg in one hour.
This is false for sure (please thank and give me brainliest)
Answer:
Abstract
Down syndrome (DS) is associated with aberrations in genetic, morphological, biochemical and physiological characteristics. A number of genes located on human chromosome 21 (HSA21) encode proteins which are thought to be involved in numerous metabolic pathways, e.g., phosphofructokinase, cystathionine β-synthase etc. Perturbations of the metabolic pathways may lead to altered drug metabolism in DS individuals. We present a review of metabolic aberrations linked to HSA21 genes in DS. We particularly focus on drug disposition, efficacy, sensitivity and toxicity of anti-leukaemic agents including methotrexate, glucocorticoids, anthracyclines and cytarabine in DS leukaemia. The different outcomes of therapy due to differential drug response, varied drug toxicity and treatment related mortality in DS leukaemia is a subject of much research and speculation. Altered drug response in DS individuals may stem from differences in pharmacokinetics, pharmacodynamics and pharmacogenetics. Further large-cohort studies in different age groups dissecting metabolic and molecular pathways involved in drug response may increase our understanding in this regard and stipulate pharmacotherapies in DS.
