Answer: False.
Explanation:
<em>The time that a person can live without feeding varies according to the age, physical condition, and health of the person</em>. There have been reports of people who spend even <u>94 days without eating</u>, and the estimated average is 40 to 70 days without eating before perishing.
Another factor to consider is the absence or presence of water. <u>Although the human being can live many days without food, without water time is limited to a few days</u>. Therefore water is more important for life.
<em>I hope this information can help you.</em>
Belly breathing
deep breathing
So, basically the question is asking what is the dose of a medication that is 2.5mg/kg.
Assuming that the child does NOT weigh 7171lbs but 71lbs; which would equal around 32kg (71 x 0.45).
So this child would have 2.5mg per 32kg. 32kg x 2.5= 80mg of medication.
Answer:
Identify the decision to start eating healthy
Explanation:
hope this helps
Answer:
Multiple sclerosis (MS) is the most common demyelinating and an autoimmune disease of the central nervous system characterized by immune-mediated myelin and axonal damage, and chronic axonal loss attributable to the absence of myelin sheaths. T cell subsets (Th1, Th2, Th17, CD8+, NKT, CD4+CD25+ T regulatory cells) and B cells are involved in this disorder, thus new MS therapies seek damage prevention by resetting multiple components of the immune system. The currently approved therapies are immunoregulatory and reduce the number and rate of lesion formation but are only partially effective. This review summarizes current understanding of the processes at issue: myelination, demyelination and remyelination—with emphasis upon myelin composition/architecture and oligodendrocyte maturation and differentiation. The translational options target oligodendrocyte protection and myelin repair in animal models and assess their relevance in human. Remyelination may be enhanced by signals that promote myelin formation and repair. The crucial question of why remyelination fails is approached is several ways by examining the role in remyelination of available MS medications and avenues being actively pursued to promote remyelination including: (i) cytokine-based immune-intervention (targeting calpain inhibition), (ii) antigen-based immunomodulation (targeting glycolipid-reactive iNKT cells and sphingoid mediated inflammation) and (iii) recombinant monoclonal antibodies-induced remyelination.Keywords: calpain, central nervous system, demyelination, fingolimod, glycolipids, lipids, multiple sclerosis, myelin, myelination, NKT cells, oligodendrocytes, remyelination, T cells
Explanation:
