Answer:
The blood pressure is directly proportional to the total length of the vessel.
Explanation:
The blood pressure is directly proportional to the total length of the vessel.
The long blood vessel will have higher the resistance and the flow of blood will be lower.
Thus, making intuitive sense, as with blood volume, as the vessel's enhanced surface area will impede blood flow. This lowering area will lead to the higher blood pressure through the blood vessel. Similarly with the reduction in the resistance of the blood vessel, the blood flow will boost.
The nurse should ask, "How much water do you drink every day?", "Do you take estrogen replacement therapy?", "Are you on steroids or other immune-suppressing drugs?".
<h3>
What is bacterial cystitis?</h3>
- The inflammation of the bladder is termed as cystitis.
- When a part of the body becomes irritated, red, or swollen it is known as inflammation.
- Urinary tract infection is the cause of cystitis. When bacteria enter the bladder or urethra and begin to multiply it causes UTI.
- The bacteria enter your urethra or bladder and cause an infection. It can also result when normally growing bacteria in your body becomes imbalanced.
To learn more about cystitis: brainly.com/question/14611917
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Answer:
Okay
Explanation:
Human topoisomerase I plays an important role in removing positive DNA supercoils that accumulate ahead of replication forks. It also is the target for camptothecin-based anticancer drugs that act by increasing levels of topoisomerase I-mediated DNA scission. Evidence suggests that cleavage events most likely to generate permanent genomic damage are those that occur ahead of DNA tracking systems. Therefore, it is important to characterize the ability of topoisomerase I to cleave positively supercoiled DNA. Results confirm that the human enzyme maintains higher levels of cleavage with positively as opposed to negatively supercoiled substrates in the absence or presence of anticancer drugs. Enhanced drug efficacy on positively supercoiled DNA is due primarily to an increase in baseline levels of cleavage. Sites of topoisomerase I-mediated DNA cleavage do not appear to be affected by supercoil geometry. However, rates of ligation are slower with positively supercoiled substrates. Finally, intercalators enhance topoisomerase I-mediated cleavage of negatively supercoiled substrates but not positively supercoiled or linear DNA. We suggest that these compounds act by altering the perceived topological state of the double helix, making underwound DNA appear to be overwound to the enzyme, and propose that these compounds be referred to as ‘topological poisons of topoisomerase I’
Answer:
D. Word roots contain the basic meaning of the
term.
Explanation:
