Answer: chloride ions
Explanation:
This movements of chloride ions into the blood plasma to replace the outward diffused bicarbonate ions is called chloride shift, It occurs to restore the blood ionic balance altered by the bicarbonate ions diffusion out into plasma
The same amounts of Chloride ions replaced the lost amount of bicarbonate ions
The answer would be c) to form images from sensory signals
The vitamin that plays an important roles in the epithelia and in the synthesis of visual pigments is vitamin A.
Vitamin A (retinol) is needed for the synthesis of photopigments. The photopigment rhodopsin is synthesized withinside the rods and is answerable for imaginative and prescient beneathneath low ranges of light. When nutritional reassets of diet A are insufficient for a protracted length of time, the quantity of visible pigment withinside the photoreceptors declines.
Vitamin A (VitA) is a micronutrient this is important for preserving imaginative and prescient, selling boom and development, and defensive epithelium and mucus integrity withinside the body.Vitamin A is critical for lots physiological processes, which includes preserving the integrity and feature of all floor tissues (epithelia): for example, the skin, the liner of the respiration tract, the gut, the bladder, the internal ear and the eye.
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Answer:
- Calcium binds to troponin C
- Troponin T moves tropomyosin and unblocks the binding sites
- Myosin heads join to the actin forming cross-bridges
- ATP turns into ADP and inorganic phosphate and releases energy
- The energy is used to impulse myofilaments slide producing a power stroke
- ADP is released and a new ATP joins the myosin heads and breaks the bindings to the actin filament
- ATP splits into ADP and phosphate, and the energy produced is accumulated in the myosin heads, starting a new cycle
- Z-bands are pulled toward each other, shortening the sarcomere and the I-band, producing muscle fiber contraction.
Explanation:
In rest, the tropomyosin inhibits the attraction strengths between myosin and actin filaments. Contraction initiates when an action potential depolarizes the inner portion of the muscle fiber. Calcium channels activate in the T tubules membrane, releasing <u>calcium into the sarcolemma.</u> At this point, tropomyosin is obstructing binding sites for myosin on the thin filament. When calcium binds to troponin C, troponin T alters the tropomyosin position by moving it and unblocking the binding sites. Myosin heads join to the uncovered actin-binding points forming cross-bridges, and while doing so, ATP turns into ADP and inorganic phosphate, which is released. Myofilaments slide impulsed by chemical energy collected in myosin heads, producing a power stroke. The power stroke initiates when the myosin cross-bridge binds to actin. As they slide, ADP molecules are released. A new ATP links to myosin heads and breaks the bindings to the actin filament. Then ATP splits into ADP and phosphate, and the energy produced is accumulated in the myosin heads, which starts a new binding cycle to actin. Finally, Z-bands are pulled toward each other, shortening the sarcomere and the I-band, producing muscle fiber contraction.
