Answer:
what should i answer lets say D
Answer:
To maintain body homeostasis
Explanation:
The endocrine system consists of the endocrine glands that release hormones directly int the blood. Those hormones bind to their receptors on the target cells and thus regulate their function. The major glands are:
pineal gland-releases melatonin, controls sleep cycle
pituitary gland-control growth, blood pressure, thyroid glands and metabolism etc.
pancreas-secretes insulin and glucagon, control of blood sugar levels
ovaries, testes-reproductive role (release female and male hormones)
thyroid gland-controls metabolism and protein synthesis
parathyroid gland-controls Ca levels
hypothalamus-connects nervous and endocrine systems, controls other glands
adrenal glands-release adrenalin and steroid hormones.
I’m not entirely sure about what you’re asking but chargaff’s rule is the thing that states that the amount of each “letter” in DNA is equal to its pair. Guanine (g) pairs with cytosine (C) and the same with adenine(A) and thymine (T). I’m sorry my answer is so vague, but I hope it helps a little!
Numerous degenerative neurological conditions, most notably Parkinson's disease, have been linked to an excessive buildup of alpha synuclein (a-syn) in the brain. Intraneuronal inclusions, often known as Lewy bodies, are neuropathological characteristics seen in Parkinson's disease, Lewy body dementia, and other synucleopathies. The aggregation of a-syn is their main structural component. A-syn accumulation, aggregation, and ensuing Lewy body formation can be attributed to a variety of biological processes. These include genetic changes in parkin, synuclein, or the deubiquitinating enzyme ubiquitin C-terminal hydrolase (UCH-L1), which results in less efficient removal of a-syn via the ubiquitin proteasomal pathway (UPP). Additionally, environmental variables and an age-related decline in antioxidant defense mechanisms that heighten oxidative stress and can have an impact on the formation or clearance of a-syn are intracellular insults.
We focused on changes in the aggregation and clearance of a-syn as impacted by the UPP and the oxidative stress pathways in our dynamic models of a-syn processing in both normal and various disease states. A free radical profile similar to that observed in vivo after exposure to the neurotoxin 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine is produced during simulation of enhanced oxidative stress (MPTP). To replicate the kinetics of a-syn that correlates to the neuropathology reported for the sporadic and hereditary types of Parkinson's disease, different model parameters of oxidative stress, UPP failure, or both routes are used. With the use of this in silico model, it is possible to evaluate the kinetics of pathway elements and more accurately identify and validate key pharmaceutical targets.
Learn more about Parkinson's disease brainly.com/question/28169444
#SPJ4
They are found in eastern australia and they usually eat Eucalyptus
