Answer:
To produce energy in form of ATP
Explanation:
The thylakoid membrane harbors photosystems that will receive light photons, ejecting electrons from a main chlorophyl molecule in a reaction center, to other acceptors. These electrons will reach the electron transport chain to create a proton gradient, and subsequently, to produce ATP. Later on these electrons will reach the other photosystem, to produce reducing power. This is in plants.
Cyclic photosystems also exist, in some bacteria, for example, and only produce energy as ATP. They also have an electron transport chain.
Inflammatory response can be defined is a reaction of the body to injury or destruction of tissues. This reaction serves to dilute, destroy or simply wall off the agent causing the injury and is localized around the area of injury.Inflammatory response takes place in the following steps:1. Tissue damage caused by injury or bacterial infection is detected.2. Vasodilators and chemotactic factors such as histamine are released.3. As a result, there is increased capillary permeability and blood flow to the area.4. There is migration of phagocytes to the site of infection.5. Phagocytes and specific serum proteins attack and destroy bacteria.<span>6. Inflammation dies down once intruder has been destroyed.</span>
Answer:
True. You can get vitamin A as "preformed vitamin A" which is already active and/or you can get vitamin A as carotenoids which the body can turn into active vitamin A.
Explanation:
There are two different types of vitamin A that can be obtained from food:
1. Preformed vitamin A, which is already active, is found in animal products such as beef, fish, poultry and dairy products.
2. Provitamin A, which is the inactive form, is found in plant-based foods, such as fruits and vegetables. The most common type of provitamin A is β-carotene, which is a carotenoid that the body can turn into active vitamin A via an enzyme named β-carotene 15,15'-monooxygenase.
Answer:
Explanation:
During transcription, DNA is converted into RNA in the nucleus of the cell.
We administered 300 mg of clindamycin intravenously at 12-h intervals for 2 days to patients with acute and chronic hepatitis, cirrhosis, and controls to determine whether clindamycin will exacerbate preexisting hepatic dysfunction or whether drug excretion will be delayed in patients with liver disease
