The hypothesis by Coleman was that the product of the ob+ gene was the appetite suppressing hormone. Hence, the homozygous ob/ob mutant are in a position to synthesize that hormone, and its circulating level would be zero. He also hypothesized that the product of the db+ gene was the receptor for the appetite-suppressing hormone. Thus, the homozygous db/db mutant would be able to synthesize the hormone but would not be in a position to respond to it. It would eat excessively and produce large amounts of body fat, which in turn would produce large amounts of appetite-suppressing hormone . In the absence of a receptor, the db/db mutant's hormone level would remain abnormally high.
Coleman's hypothesis were confirmed when the precise functions of the ob+ and db+ genes were determined. The peptide hormone encoded by the ob+ gene was named Leptin.
Damage to nerve or bone can happen when you puncture infant's extremities to collect capillary blood. The safest region to do this is the lateral side of <span>the infant's heel. With this method, the chance damage to the nerve of bone is much less than the middle of an infant or the finger.</span>
Answer:
The agent causing the pneumonia, where bluish-green pus was found, is most likely Pseudomonas aeruginosa.
Explanation:
Pseudomona aeuriginosa is a gram-negative bacteria that is one of the main causes of hospital-acquired infections, including pneumonias in mechanically ventilated patients.
One of the characteristics of P. aeuriginosa is the formation of a bluish-green pus, since it has the capacity to form cyanide-based blue pigment upon contact with the organic tissues it infects. This is the reason why previously P. aeruginosa was called a pyocyanic bacillus.
<em> The other options are not correct because the only bacterium that produces blue-green pus is P. aeruginosa.</em>
The answer is density-dependent factor
