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<u>6</u><u>2</u><u>.</u><u> </u><u>B</u><u>.</u><u> </u><u>2</u><u> </u><u>AND</u><u> </u><u>4</u></h2>
<u>HOP</u><u>E</u><u> IT</u><u> </u><u>HELPS</u><u> </u><u>YOU</u>
<span>There are numerous proteins in muscle. The main two are thin actin filaments and thick myosin filaments. Thin filaments form a scaffold that thick filaments crawl up. There are many regulatory proteins such as troponin I, troponin C, and tropomyosin. There are also proteins that stabilize the cells and anchor the filaments to other cellular structures. A prime example of this is dystrophin. This protein is thought to stabilize the cell membrane during contraction and prevent it from breaking. Those who lack completely lack dystrophin have a disorder known as Duchene muscular dystrophy. This disease is characterized by muscle wasting begininng in at a young age and usually results in death by the mid 20s. The sarcomere is the repeating unit of skeletal muscle.
Muscle cells contract by interactions of myosin heads on thick filament with actin monomers on thin filament. The myosin heads bind tightly to actin monomers until ATP binds to the myosin. This causes the release of the myosin head, which subsequently swings foward and associates with an actin monomer further up the thin filament. Hydrolysis and of ATP and the release of ADP and a phosphate allows the mysosin head to pull the thick filament up the thin filament. There are roughly 500 myosin heads on each thick filament and when they repeatedly move up the thin filament, the muscle contracts. There are many regulatory proteins of this contraction. For example, troponin I, troponin C, and tropomyosin form a regulatory switch that blocks myosin heads from binding to actin monomers until a nerve impulse stimulates an influx of calcium. This causes the switch to allow the myosin to bind to the actin and allows the muscle to contract. </span><span>
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Hormones are chemical substances that help to regulate processes in the body. Hormones are secreted by glands and travel to their target organs in the bloodstream. Hormones can be used to control human fertility.
Answer:
Permanent slides ensures preservation of the body parts of the tiny aquatic animals and also also aids good visualization.Most of the exoskeleton of these tiny animals are lost during temporary slides preparation, thus loss of visualization.
Because the animals are transfixed to a point, their body parts can not be moved.
It also saves time, energy and resources spent during observation.
However,because of dehydration during preparation their natural color may be lost which may affect observation of natural features
Explanation:
Answer:
Indivisuals with producing an abnormal form of extracellular protein fibrillin are suffering from Marfan syndrome ehich is caused by genetic mutation in the FBN1 gene.
Explanation:
Gene mutations in FBN1 gene results in the production of an abnormal extracellular matrix fibrillin-1 protein that cannot function properly. These gene mutations basically reduce the amount of fibrillin-1 produced by the cell, alter the structure of fibrillin-1, or causes the impairment of the transport of fibrillin-1 out of the cell.
As a result, protein is poorly incorporated into extracellular matrix. Hence, indivisuals with Marfan syndrome present following symptoms
Tall stature.
Disproportionately long arms, legs and fingers.
Sternum either protrudes outward or dips inward.
Arched palate and crowded teeth.
Heart murmurs.
Extreme nearsightedness.