Answer:
you can tell the benefits of healthy food and you can tell the benefits of junk food also you can tell that you eat healthy food and it provides us energy
The priority education topics to teach the patient which has a history of gastroesophageal reflux disease include;
- Remaining in upright position at least 2 hours after meals.
- Proper mastication of food and eating slowly.
<h3>What is GERD?</h3>
This is referred to as Gastroesophageal reflux disease which is characterized by bile and stomach acid irritating the food pipe lining due to the repeated back flow to the area.
The topics which should be taught is eating slowly and proper mastication so as to ensure that digestion occurs faster and sitting upright will also assists to prevent the back flow of the acids and enzymes.
Read more about Gastroesophageal reflux disease here brainly.com/question/3006031
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Answer:
A. Malnutrition is causing symptoms that mimic NCD
Explanation:
Malnutrition can happens if a person body doesn't get the necessary nutrients needed due to poor diet which in turn leads to deficiencies and imbalances in the person's intake of nutrients.
Causes of MALNUTRITION can include inappropriate dietary choices, difficulty obtaining food as well as physical and mental health conditions in which such person may begin to have in delayed growth or low weight.
MALNUTRITION can sometimes cause symptoms that mimic NCD which non-communicable disease that is not transmissible from one person to another such as fatigue, dizziness and weight loss.
Therefore based on the information given Dwayne's is most likely to be diagnosis with MALNUTRITION THAT IS CAUSING SYMPTOMS THAT MIMIC NDC.
Local anesthetics inhibit nerve conduction in a reversible manner without altering the nerve. The inhibition appears rapidly and for a longer or shorter duration depending on the products and the concentrations used. The extent of the territory rendered insensitive to pain depends on the modes of administration of the local anesthetic, either at the level of the nerve endings, or at the level of a nervous trunk, for example.
They act at the level of the neuronal membrane by interfering with the process of excitation and conduction. The anesthetic crosses the axon membrane, rich in lipids, in the form of base before taking up a cationic form on the internal face of the neuron where the pH is more acidic.
At this level, there is a blockage of nerve conduction by decreasing the membrane permeability to sodium ions that occurs during the depolarization phase. As the progression of the anesthetic action along the nerve increases, the threshold of excitability increases and the conduction time increases. This is completely blocked from a certain concentration of local anesthetic.
The nerve fibers are unequally sensitive to the action of local anesthetics: they disappear in order: the painful, thermal, tactile sensations.
