<span>As
I know Gregor Mendel - Austrian naturalist, botanist and a religious leader, a
monk, founder of the doctrine of heredity (Mendelism). By applying statistical
methods for the analysis of results of the hybridization of pea varieties,
formulated the laws of heredity. In 1856, Mendel began his experiments in
crossing different varieties of peas, differing in a single, strictly defined
criteria (for example, the shape and color of seeds). Precise quantitative
account of all types of hybrids and statistical processing of the results of
experiments that he conducted for 10 years, allowed him to formulate the basic
laws of heredity - the splitting and combining of hereditary
"factors". Mendel showed that these factors are separated and not
crossing merge and disappear. Although the crossing of two organisms with
contrasting features (for example, yellow or green seeds) in the next
generation of hybrids appears only one of them.</span>
Answer:
we get chemical energy from food
Explanation:
There is chemical energy in food so when you eat food you get chemical energy from it.
The first geneticist has identified an obese allele that he or she believes to be recessive. We will define his or her allele as o1 and the normal allele as O1. The obese allele appears to be recessive based on the series of crosses he or she performed.
Cross 1 with possible genotype:
Obese (o1o1) × Normal (O1O1) F1 All normal (O1o1)
Cross 2 with possible genotypes:
F1 normal (Oo1) × F1 normal (O1o1) F2 8 normal (O1O1 and O1o1)
2 obese (o1o1)
Cross 3 with possible genotypes:
Obese (o1o1) × Obese (o1o1) All Obese (o1o1)
A second geneticist also finds an obese mouse in her colony and performs the same types of crosses, which indicate to her that the obese allele is recessive. We will define her obese allele as o2 and the normal allele as O2.
The cross of obese mice between the two different laboratories produced only normal mice. These different alleles are both recessive. However, they are located at different gene loci. Essentially, the obese mice from the different labs have separate obesity genes that are independent of one another.
The likely genotypes of the obese mice are as follows:
Obese mouse 1 (o1o1O2O2) × Obese mouse 2 (O1O1o2o2)
F1 All normal (O1o1O2o2)
Healthy working out and sleeping brain cells also
