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Anna35 [415]
3 years ago
14

However, when the proteins formed by the genes have their amino acids sequenced, there is no difference observed between the two

. What is the most likely explanation for this observation?
Biology
1 answer:
Musya8 [376]3 years ago
8 0

Answer:

The correct answer is: Variation in nucleotide sequences are located in those regions of the gene which do not affect the sequence of the protein due to genetic code degeneracy.

Explanation:

  • Proteins are encoded from the genes located in the chromosomes.
  • The genes are made up of the heritable DNA (Deoxyribonucleic Acid) sequences.
  • These genes are transcribed by the enzyme called RNA polymerase into mRNA (messenger Ribonucleic Acid) sequences in the nucleus.
  • The mRNA is then translated into protein sequences by the Ribosome in the cytoplasm or the RER (Rough Endoplasmic Reticulum).
  • The Ribosome reads the mRNA code in form of triplets, that is, three consecutive nucleotide are read as a single code that encode for a single amino acid. This triplet code that responsible for coding for a single amino acid is called a Codon.
  • The Codons are degenerate in nature. This means that among the three nucleotide in a codon the first two nucleotide is specific for a particular amino acid. The third nucleotide is "wobble" in nature. This means that whatever may be the nucleotide in the third position it will not change the amino acid coded by the first two nucleotide.
  • Hence, a single amino acid is coded by more than one codon.
  • So, gene sequences showing variation in the wobble nucleotide position will encode for the same protein.
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Generally, a pulse pressure should be at least 25 percent of the systolic pressure. A pulse pressure below this level is described as low or narrow. This may occur, for example, in patients with a low stroke volume, which may be seen in congestive heart failure, stenosis of the aortic valve, or significant blood loss following trauma. In contrast, a high or wide pulse pressure is common in healthy people following strenuous exercise, when their resting pulse pressure of 30–40 mm Hg may increase temporarily to 100 mm Hg as stroke volume increases. A persistently high pulse pressure at or above 100 mm Hg may indicate excessive resistance in the arteries and can be caused by a variety of disorders. Chronic high resting pulse pressures can degrade the heart, brain, and kidneys, and warrant medical treatment.

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After blood is ejected from the heart, elastic fibers in the arteries help maintain a high-pressure gradient as they expand to accommodate the blood, then recoil. This expansion and recoiling effect, known as the pulse, can be palpated manually or measured electronically. Although the effect diminishes over distance from the heart, elements of the systolic and diastolic components of the pulse are still evident down to the level of the arterioles.

This image shows the pulse points in a woman’s body.

Figure 2. The pulse is most readily measured at the radial artery, but can be measured at any of the pulse points shown.

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Pulse can be palpated manually by placing the tips of the fingers across an artery that runs close to the body surface and pressing lightly. While this procedure is normally performed using the radial artery in the wrist or the common carotid artery in the neck, any superficial artery that can be palpated may be used. Common sites to find a pulse include temporal and facial arteries in the head, brachial arteries in the upper arm, femoral arteries in the thigh, popliteal arteries behind the knees, posterior tibial arteries near the medial tarsal regions, and dorsalis pedis arteries in the feet. A variety of commercial electronic devices are also available to measure pulse.

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