Answer:
<u>b. synthesizes new DNA only in the 5' to 3' direction.</u>
Explanation:
DNA Polymerase III is involved in replication of DNA, it is involved in the proofreading of the replication mistakes by activity reading 3' to 5' and synthesizing 5' to 3' . It can add nucleotides to the strand at the rate of about 1000 nucleotides per second.
Answer:
The parents are of genotype heterozygous dominant and homozygous recessive. Supposing the dominant allele is N and the recessive allele is n, one of the parents will be Nn while the other nn. The phenotypic (based on visible characteristics) ratio will be 1:1 for dom/rec and rec/rec
Explanation:
Http://www.pbs.org/wgbh/nova/cancer/folkman.html
I believe this website will help :)
Long-term potentiation (LTP) is considered a cellular correlate of learning and memory. The presence of G protein-activated inwardly rectifying K(+) (GIRK) channels near excitatory synapses on dendritic spines suggests their possible involvement in synaptic plasticity. However, whether activity-dependent regulation of channels affects excitatory synaptic plasticity is unknown. In a companion article we have reported activity-dependent regulation of GIRK channel density in cultured hippocampal neurons that requires activity oF receptors (NMDAR) and protein phosphatase-1 (PP1) and takes place within 15 min. In this study, we performed whole-cell recordings of cultured hippocampal neurons and found that NMDAR activation increases basal GIRK current and GIRK channel activation mediated by adenosine A(1) receptors, but not GABA(B) receptors. Given the similar involvement of NMDARs, adenosine receptors, and PP1 in depotentiation of LTP caused by low-frequency stimulation that immediately follows LTP-inducing high-frequency stimulation, we wondered whether NMDAR-induced increase in GIRK channel surface density and current may contribute to the molecular mechanisms underlying this specific depotentiation. Remarkably, GIRK2 null mutation or GIRK channel blockade abolishes depotentiation of LTP, demonstrating that GIRK channels are critical for depotentiation, one form of excitatory synaptic plasticity.
Learn more about receptors here:
brainly.com/question/11985070
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The nucleus is what houses the cells genetic information. Hope this helps!
