I would assume that centipedes would die if you cut them in half because you would cause them to bleed out and cut off some vital organs from the brain.
Mutations present permanent and heritable changes in the genome. We distinguish gene and chromosome mutations. Mutations in the level of genes are called point mutations because only one to several nucleotides are changed. These processes are known as deletion, insertion, and substitution. Deletion presents a loss of nucleotides which changes DNA sequence. When new nucleotides are embedded in the DNA chain, it is known as insertion. Substitution implies the process where new nucleotides are inserted while the ones that were present in that specific spot in a DNA molecule are deleted.
Mutations can also appear in chromosomes altering their number and structure. There are four types of mutations - deletion, duplication, inversion, and translocation. In deletion, a whole or one part of a chromosome is lost. Duplication presents an extra copy of a whole or one part of a chromosome. In an inversion, parts of a chromosome change order, while in translocation a part of one chromosome detaches and then connects to another.
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Location 2 is cooler than Location 3 and Location 1 because it's at a higher altitude
<span>The pancreas secretes digestive enzymes in the slender instincts, these enzymes produce endocrine secretions through the hormones and pass through the blood. Endocrine cells called pancreatic islets secrete insulin and glucagon and have endocrine functions that are formed through the hormones and pass through the bloodstream.</span>
<span>Lafora disease is the most severe teenage-onset progressive epilepsy, a unique form of glycogenosis with perikaryal accumulation of an abnormal form of glycogen, and a neurodegenerative disorder exhibiting an unusual generalized organellar disintegration. The disease is caused by mutations of the EPM2A gene, which encodes two isoforms of the laforin protein tyrosine phosphatase, having alternate carboxyl termini, one localized in the cytoplasm (endoplasmic reticulum) and the other in the nucleus. To date, all documented disease mutations, including the knockout mouse model deletion, have been in the segment of the protein common to both isoforms. It is therefore not known whether dysfunction of the cytoplasmic, nuclear, or both isoforms leads to the disease. In the present work, we identify six novel mutations, one of which, c.950insT (Q319fs), is the first mutation specific to the cytoplasmic laforin isoform, implicating this isoform in disease pathogenesis. To confirm this mutation's deleterious effect on laforin, we studied the resultant protein's subcellular localization and function and show a drastic reduction in its phosphatase activity, despite maintenance of its location at the endoplasmic reticulum.
I got my information from </span>https://www.ncbi.nlm.nih.gov/pubmed/14722920