Answer: Saturated
Explanation: Good luck! :D
Answer:
Option (3)
Explanation:
Unconformity is defined as the gaps in the sequence of geological rocks. It is a geological contact that marks the boundary between the different types of rocks. In simple words, they are also known as the time of non-deposition. These unconformities have great significance in the field of geology. They are of 4 types, namely-
(1) Angular unconformity- The sedimentary rocks are initially formed and deposited in an area, after that the area is tilted and erosion takes place. Now, the new sediments are deposited forming an angle, which is commonly known as an angular unconformity.
(2) Non-conformity- When the sedimentary rocks are deposited over the igneous or metamorphic rocks, then it is known as the non-conformity.
(3) Para conformity- When the sedimentary rocks are deposited parallel to the ground surface, and absence of erosion activity, then these surface appears to be a normal (simple) bedding plane. This is known as the para conformity.
(4) Disconformity- When the sedimentary rocks are formed and undergoes erosion, then new sedimentary rocks are deposited over this and diminishes the unconformity. This forms a discontinuity.
Thus, the unconformity arises due to both erosion and lack of rock deposition.
Hence, the correct answer is option (3).
Answer: A procedure in which stain is applied until the desired intensity of tissue coloration is attained
Explanation:
Answer:
Thanks for you question. Your hypothesis suggests a linear relationship between serum Cholesterol levels and MI. This hypothesis seems to ignore the difference in the prevalence and effectiveness of LDL receptors in the FH patient.
FH patients who have inherited the mutation from both parents have very few LDL receptors in their blood and therefore almost no ability to pass the unused Cholesterol through the liver. FH patients who are heterozygous will have more LDL receptors although both will find Cholesterol removal problematic without the addition of a PCSK9 inhibitor.
In short, your hypothesis need to account for other factors that are in play.
Explanation:
Consider my case. I am a 64 year old male who has Heterozygous Familial Hypercholesterolemia. Before treatment at age 12 my Total cholesterol was 510 mg/dl. My genetic testing shows two mutations to the LDL Receptor gene with only one mutation being pathogenic. My first heart attack was at 47 and first stroke at 62. My current LDL is too low to detect with the use of a PCSK9 inhibitor (Repatha®).
