There will be to much sugar to drink from the water or you can and go to the hospital.
Oh no I didn’t lol I didn’t get a picture from my daughter so she was going back lol I got a little crazy girl I didn’t want her lol I was just kidding I didn’t want her lol I was going crazy and I was like crazy crazy oh yeah I know it’s not that hard to say but I’m going crazy crazy and I can’t sleep now tyyyyyyy I just want my sleep lol I’m good I just
Answer:
Cellular respiration is an important metabolic process that moves sulfur from living organisms into the atmosphere.
Explanation:
Cellular respiration is mainly based in the carbon cycle, because most of the compounds involved have carbon in it structure.
sulfur can release to the atmosphere by burning fossil fuels or by volcanic eruptions in the form of SO2, this is a normal gas, despite is a toxic gas, but the quantities that are formed are too little.
Like a cycle, the animals can obtain sulfur if they eat plants because the plants take from the soil the sulfur and transformed it into minerals like sulfates.
Like a cycle, the element sulfur just change from one substance to another.
Answer:
Golgi apparatus and extracellular matrix
Explanation:
The Golgi complex lies at the heart of the secretory pathway and is responsible for modifying proteins and lipids, as well as sorting newly synthesized molecules to their correct destination. As a consequence of these important roles, any changes in its proteome can negatively affect its function and in turn lead to disease.
Disruption of specific transport steps between the endoplasmic reticulum (ER), Golgi complex, endosomal-lysosomal system and the plasma membrane all can have dramatic consequences on the cell, and increasingly defects in the molecular machinery regulating membrane traffic are being linked to hereditary diseases.
Any changes to the proteome of the Golgi complex would affect its homeostasis and consequently the flux of proteins trafficking through it.
One possible consequence of mutations in Golgi complex proteins is that they cause gross changes in the morphology of the entire organelle and protein mislocalization, which together result in functional problems such as impairment of glycosylation.
Genetic studies of humans and mice continue to highlight the nonredundant mechanical role of components in complexes that anchor cells to extracellular matrices. At the same time, recent data provide exciting insights into, critical roles of transcription factors in regulating differentiation and function of matrix-producing cells.
