Answer:
Mutations in introns create alternative splicing site which in turn make mRNA with reduced or no translation.
Explanation:
Introns regulate the splicing of heterogenous RNA during post transcriptional modifications as well as affect the stability of mature mRNA. The stable mRNA is more likely to be translated into proteins. So, introns also regulate the translation of mRNA.
In human beta thalassemia, mutations in intron create alternative splicing sites which in turn affect the formation of beta globin chain of hemoglobin. Lack of beta globin chain reduces the amount of functional hemoglobin and causes anemia.
I would say the results of the experiment but not the hypothesis, if we're saying possible corrections to the hypothesis count as something outside of it
Organelle near the nucleus in animal cells
