Answer: Creo que es B
Explanation:
P.S(eso fue muy difícil. Lo siento mucho si se equivoca.)
Which species are you talking about
Answer:
When seen on a Wright-stained peripheral blood film, a young red cell that has just extruded (lost its) nucleus is referred to as a polychromatophilic cell.
Explanation:
On Wright-stained smears, slightly immature red cells that do not have nuclei (reticulocyte stage) look blue-gray because they still have some ribonucleic acid in them (RNA). These cells are commonly referred to as polychromatophilic cells. Most of the time, polychromatophilic cells are bigger than mature red cells, and their blue-gray color makes them different from macrocytes. Polychromatophilic red cells also tend to lack the central pallor.
When the remaining mRNA and ribosomes are stained with supravital dyes, they make the red cells look like a "reticular" mesh network. This is how the name "reticulocyte" came about. It is to be noted that not all reticulocytes show up as polychromatophils when stained with Wright-Giemsa.
Learn more about peripheral blood smear here:
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The question is incomplete, so the complete question is as follows:
A chloroplast has stopped producing ATP and NADPH. Which of the following is most likely causing this?
a.The chloroplast has used up its supply of chlorophyll molecules.
b.Electrons have stopped moving through the electron transport chain.
c.The sun has risen and the chloroplast now can make more chlorophyll.
Answer:
b.Electrons have stopped moving through the electron transport chain.
Explanation:
ATP and NADPH are produced in the light dependent reaction stage of the photosynthesis that occurs within chloroplast grana.
ATP and NADPH are produced due to the movement of electron and proton (H+) in the electron transport chain, so if the movement of electrons will stopped in the electron transport chain, the formation of ATP and NADPH will also get stop.
Hence, the correct answer is "b".
Answer and explanation;
-The insulin receptor itself has decreased kinase activity leading to fewer binding sites for singling effectors to become activated.
Therefore; the signaling effectors are less likely to be activated.
-Additionally,, feedback inhibition decreases signaling effector binding to receptor.
-The lack of activation of these initial signaling effectors prevents any subsequent steps from happening.
-We can thus conclude this by saying that higher insulin concentrations are necessary to obtain the same level of signaling leading to physiological effects that would exist in someone without type-2 diabetes.
-Also the requirement for higher insulin concentration means that the cells that respond to insulin are resistant to its effect.