Answer:
directional selection
Explanation:
Directional selection is the most common type of natural selection and occurs when some individuals with characteristics favorable to the conditions of the environment in which they live, have survival advantages over individuals who do not have this advantage, who end up dying.
Imagine, for example, a graph showing the directional selection in the same species of moths. Moths of the same species have white and brown collations, in summer, brown moths can camouflage themselves on tree trunks, while white moths cannot and are easily captured by their predators, which means that the amount of white moths decrease. In this graph, the population of white moths would be at a minimum, at the same time that the population of brown moths would be at maximum.
However, with the arrival of the reverse, snow begins to cover the trees, allowing white moths to camouflage themselves more easily. The brown moths, then, are very exposed to predators, causing their population to reach the minimum while the population of white moths reaches the maximum.
Answer:
The cuticle is the hair's protective layer, the cortex forms the pigment of the hair, the health of the cortex depends on the cuticle protecting it while the medulla is the innermost of the hair.
Explanation:
The cuticle is the hair outermost layer, the cortex is the middle structure for strength while medulla is the inner most.
Answer:
i need more details to answer.
Explanation:
sorry
Answer:
- Calcium binds to troponin C
- Troponin T moves tropomyosin and unblocks the binding sites
- Myosin heads join to the actin forming cross-bridges
- ATP turns into ADP and inorganic phosphate and releases energy
- The energy is used to impulse myofilaments slide producing a power stroke
- ADP is released and a new ATP joins the myosin heads and breaks the bindings to the actin filament
- ATP splits into ADP and phosphate, and the energy produced is accumulated in the myosin heads, starting a new cycle
- Z-bands are pulled toward each other, shortening the sarcomere and the I-band, producing muscle fiber contraction.
Explanation:
In rest, the tropomyosin inhibits the attraction strengths between myosin and actin filaments. Contraction initiates when an action potential depolarizes the inner portion of the muscle fiber. Calcium channels activate in the T tubules membrane, releasing <u>calcium into the sarcolemma.</u> At this point, tropomyosin is obstructing binding sites for myosin on the thin filament. When calcium binds to troponin C, troponin T alters the tropomyosin position by moving it and unblocking the binding sites. Myosin heads join to the uncovered actin-binding points forming cross-bridges, and while doing so, ATP turns into ADP and inorganic phosphate, which is released. Myofilaments slide impulsed by chemical energy collected in myosin heads, producing a power stroke. The power stroke initiates when the myosin cross-bridge binds to actin. As they slide, ADP molecules are released. A new ATP links to myosin heads and breaks the bindings to the actin filament. Then ATP splits into ADP and phosphate, and the energy produced is accumulated in the myosin heads, which starts a new binding cycle to actin. Finally, Z-bands are pulled toward each other, shortening the sarcomere and the I-band, producing muscle fiber contraction.
