This would be Newton's first law of motion as it is a factual statement based on observations.
The type of shock presented by a patient with a known history of gastric ulcers and gastrointestinal bleeding with recent history of passing a lot of blood is called hypovolemic shock. The patient is now on the later stages of shock as he is tachycardic and hypotensive (significant as mechanisms to maintain a normal blood pressure is failing at this point).
The steps of fracture repair include:
- Hematoma formation.
- Fibrocartilage Callus Formation.
- Bony Callus Formation.
- Bone remodeling.
<h3>What is a fracture?</h3>
A fracture is a complete or partial break in a bone. The causes of bone fractures include trauma, overuse, and diseases that weaken bones.
In this case, Kyndall was lucky because the fracture occurred about 3 inches below the epiphyseal plate as this means the fracture does not break through the bone at the growth plate.
An improperly treated growth plate fracture could result in a fractured bone.
Lastly, the type of fracture is a nondisplaced fracture is one in which the bone cracks or breaks but retains its proper alignment.
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The answer is; glycolysis
This process converts glucose molecule to pyruvate. It is an oxygen-independent pathway, unlike the Krebs cycle. Glycolysis occurs in the cell cytoplasm while the Krebs cycle (aerobic pathway) occurs in the mitochondria. In the presence of oxygen, the product of glycolysis, i.e pyruvate, is fed to the Krebs cycle. If oxygen is unavailable the pyruvate is converted to lactate.
Answer:
CHK2, p53, p21, cell cycle progression.
Explanation:
In a normal cell with no LFS mutation, during the G1 stage of cell cycle progression, the CHK2 activates if there is damage in the DNA. CHK2 activates p53, which is a tumor suppressor protein that will hold the cell cycle in G1/S until the DNA is repaired. The p53 protein activates p21, a protein that binds to CDK2 and stops the cell cycle. The cell cycle will continue once the damage is repaired.
