124 gtt/min drops per minute should the nurse regulate the infusion . Because Sodium nitroprusside (nipride) 50 mg is mixed in d5w 250 ml.
182/2.2 Equals 82.73 kg when converting from lbs to kg. For this client, determine the dosage: 413.65 mcg/min = 5 mcg x 82.73. Calculate how much mcg Sodium nitroprusside are present in 1 ml: 200 mcg per ml is 250/50,000 mcg.
The customer is to receive 2.07ml per minute (413.65 mcg/min x 200 mcg/ml), or 413.65 mcg/min x 200 mcg/ml. When the drip factor is 60 gtt/ml, the equation is 60 2.07 = 124.28 gtt/min OR, when utilising dimensional analysis, the equation is 60 gtt/ml X 250 ml/50 mg X 1 mg/1,000 mcg X 5 mcg/kg/min X 1 kg/2.2 pounds X 182 lbs.
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In the given scenario, icd-10-cm code reported is H02.423.
What is icd-10-cm code?
The ICD-10-CM is a morbidity classification developed by the United States that is used to classify diagnoses and reasons for visits in all health care settings.
Upper eyelid drooping is caused by a muscle disorder (myogenic). Look for Ptosis/eyelid in the ICD-10-CM Alphabetical Index, which states to see Blepharoptosis.
Look for Blepharoptosis and you'll be directed to H02.423, where the sixth character indicates laterality.
The sixth character of three stands for bilateral. There is only one code for both eyelids, not two separate codes.
This, this should be the icd-10-cm code.
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Answer:
When he realized that Marcee, the imaginary niece of his imaginary roommate never gets any older.
Answer:
Okay
Explanation:
Human topoisomerase I plays an important role in removing positive DNA supercoils that accumulate ahead of replication forks. It also is the target for camptothecin-based anticancer drugs that act by increasing levels of topoisomerase I-mediated DNA scission. Evidence suggests that cleavage events most likely to generate permanent genomic damage are those that occur ahead of DNA tracking systems. Therefore, it is important to characterize the ability of topoisomerase I to cleave positively supercoiled DNA. Results confirm that the human enzyme maintains higher levels of cleavage with positively as opposed to negatively supercoiled substrates in the absence or presence of anticancer drugs. Enhanced drug efficacy on positively supercoiled DNA is due primarily to an increase in baseline levels of cleavage. Sites of topoisomerase I-mediated DNA cleavage do not appear to be affected by supercoil geometry. However, rates of ligation are slower with positively supercoiled substrates. Finally, intercalators enhance topoisomerase I-mediated cleavage of negatively supercoiled substrates but not positively supercoiled or linear DNA. We suggest that these compounds act by altering the perceived topological state of the double helix, making underwound DNA appear to be overwound to the enzyme, and propose that these compounds be referred to as ‘topological poisons of topoisomerase I’
