Answer:
This is due to the event of Speciation that happened for the rodents in Island B but not for the rodents in Island C.
Explanation:
- Due to splitting of the population,
- The sub-population of rodents formed in Island B are B1 and B2.
- The sub-population of rodents formed in Island C are C1 and C2.
- In case of Island B, each of the B1 and B2 sub-populations that got split from each other developed certain mutations that were necessary for them to adapt to the particular diverse environment each of them were exposed to, through the period of 50,000 years. These mutations were so varied that reproductive isolation was generated between them that resulted in each of them to develop into different species.Hence, speciation happens here and B1 and B2 are incapable of inter-breeding.
- In case of Island C, each of the C1 and C2 sub-populations that got split might have got exposed to similar environmental change or no environmental change or the environmental change might have been too small to cause drastic change in each of the sub-populations. As a result of this the two sub-populations might have acquired certain mutations to adapt to the environment each of them were exposed to, through a period of 100,000 years. These mutations might not have been too variable or contrasting to cause reproductive isolation between C1 and C2. Hence, no new speciation happens here and C1 and C2 are capable of inter-breeding.
Well the more that get cut down, the less oxygen we get, aswell as more carbon in the atmosphere because there is less trees to absorb it.
Answer:
d
Explanation:
it takes the weight of that much ice to cause the plastic to flow.
<span>Rhabdomyolysis constitutes a common cause of acute renal failure and presents paramount interest. A large variety of causes with different pathogenetic mechanisms can involve skeletal muscles resulting in rhabdomyolysis with or without acute renal failure. Crush syndrome, one of the most common causes of rhabdomyolysis presents increased clinical interest, particularly in areas often involved by earthquakes, such as Greece and Turkey. Drug abusers are another sensitive group of young patients prone to rhabdomyolysis, which attracts the clinical interest of a variety of medical specialties.
We herein review the evidence extracted from updated literature concerning the data related to pathogenetic mechanisms and pathophysiology as well as the management of this interesting syndrome.
Keywords: Rhabdomyolysis, acute renal failure, myoglobin, crush syndrome
The first case of the crush syndrome, which constitutes one of the main causes of rhabdomyolysis, was reported in Sicily in 1908, after an earthquake1,2. In 1930, in the Baltic area, an epidemic of myoglobinuria was observed due to consumption of contaminated fish. Interest in rhabdomyolysis and crash syndrome was stimulated during the World War II particularly after the bombing in London, where the victims developed acute renal failure and myoglobinuria1.
Rhabdomyolysis is a rupture (lysis) of skeletal muscles due to drugs, toxins, inherited disorders, infections, trauma and compression3. Lysis of muscle cells releases toxic intracellular components in the systemic circulation which leads to electrolyte disturbances, hypovolemia, metabolic acidocis, coagulation defects and acute renal failure due to myoglobin4.
The skeletal muscle consists of cylindrical myofibrils, which contain variant structural and contraction proteins. Actin and myosin, arranged in thin and thick filaments respectively, form the repeated functional units of contraction, the sarcomeres5. The sarcoplasmic reticulum constitutes an important cellular calcium storage. It is structurally connected to the t-tubules, that are formed by invaginations of the muscle cell plasma membrane, the sarcelemma, around every fibril (Figure 1). After the sarcelemma depolarization, the stimulation arrives, through the t-tubules junctions, at the sarcoplasmic reticulum, inducing the calcium ions release and triggering muscle contraction6.</span>
