Answer:
Similarities: both are chemical messengers, both can have (but don't necessarily have) similar structures (made from amino acids), both can be produced by the central nervous system (hypothalamus produces some hormones), certain molecules can act as both hormones and neurotransmitters (example: norepinephrine).
Differences: neurotransmitters are released by neurons across a synaptic gap, have very short distances to travel, and act very quickly on the neighboring cell. Hormones are released by glands and often have to travel longer distances, which means that they are slower-acting. Also, hormones are typically used for regulation (negative feedback) while neurotransmitters are used for stimulation of a neighboring cell.
Explanation:
Answer: hi, im here to help :3
so, you can either use plants, use pesticides and fertilizers less often or the one thing i know is consider a rain barrel.
Answer:
True
Explanation:
Some deviations from normal homeostasis activate the positive feedback loops to control the conditions which are otherwise regulated by negative feedback mechanisms.
For example, the blood levels of respiratory gases and H+ ions are regulated by a negative feedback system via chemoreceptors. The increased partial pressure of carbon dioxide gas and lowered pH or lowered partial pressure of oxygen in the blood are sense by central and peripheral chemoreceptors which in turn activate the neurons of the dorsal respiratory group (DRG).
The activated DRG triggers an increased in the rate and depth of the breathing to facilitate the inhalation of more oxygen and exhalation of CO2 to restore the normal levels.
However, hypocapnia inactivates the chemoreceptors and does not allow negative feedback to restore the normal CO2 levels in the blood.
Under such conditions, the positive feedback loop stimulates the DRG neurons more strongly in response to the increased partial pressure of CO2 above the normal levels than when the partial pressure of oxygen falls below the normal level. These dangerously lowered oxygen levels may also cause fainting.
It can be expected that there
will be closure of the patent ductus arteriosus for this is the effect of
indomethacin. The adverse effect would include platelet dysfunction, decrease
gasto-intestinal motility and an increase in necrotizing enterocolitis. With this,
the nurse should anticipate the possible outcomes where there will be increase
bleeding time and decrease gastro-intestinal function after giving
indomethacin.