YES YES YES YES YES YES YES YES I don’t get what u are saying but points for me!
Answer:
Muscle contraction function.
Explanation:
The nerve endings possess synaptic acetylcholine vesicles ready to be released. The action potential depolarizes the presynaptic terminal and increases the concentration of axoplasmic calcium; Acetylcholine molecules are thus released, so that the concentration of the neurotransmitter at postsynaptic (nicotinic) receptors is temporarily increased. This is followed by post-synaptic membrane depolarization, muscle membrane action potential with increased rnioplasmic calcium concentration, and finally muscle contraction. Acetylcholine is hydrolyzed by acetylcholinesterase and resynaptic at the presynaptic level by cholinecetyltransferase. The etiopathogenesis of myasthenia gravis is autoimmune and there are antibodies against acetylcholine receptors that circulate in the blood, as well as a decrease in the number of receptors on the motor plates, that is, it is produced by the postsynaptic blockage of the myoneural plaque, that generates fatigue and localized or generalized muscle weakness that is characterized by the worsening of the contractile force of the muscle.
➜ <u>Oxyhaemoglobin</u>.
➜ <u>Carboxyhaemoglobin</u> and as carbonic acid (CO2 dissolved in blood plasma).
Afunction of steroids is to increase muscle mass to help you get an advantage on your competitors.
Answer:
Transcription of gene A is not affected
Explanation:
A transcriptional repressor is a DNA/RNA-binding protein that acts to suppress gene expression and/or protein synthesis by binding to target sequences. Moreover, a silencer is a DNA region that binds to particular transcriptional repressors in order to suppress gene expression. In this case, the transcriptional repressor is activated by binding to an effector molecule X (e.g., another protein). In consequence, and since the activated repressor-X complex is not more able to bind the silencer of gene A, it is expected that such complex will not be able to affect transcription of the target gene.
