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Readme [11.4K]
3 years ago
7

The brightest star in the universe, a supernova, is thought to be about a million times brighter than our own Sun. If Polaris, l

ocated about 434 light years from Earth, were to go supernova today, why would no one currently on Earth ever know it happened?
A) It would take the light 434 years to reach the Earth.

B) The light would be so dim that our eyes would not detect it.

C) The light would be so bright that or eyes could not detect it.

D) The light from the supernova would never reach the Earth as it would travel in a different direction.
PLZ HURRY I NEED THIS ANSWER

Biology
1 answer:
NemiM [27]3 years ago
7 0

If Polaris were to go supernova today, no one currently on Earth ever know it happened because it would take 434 light years to reach the Earth

Explanation:

The Polaris or the North Star that we can see at the North Pole of the Earth in the night historically dates back to 434 years in the past. If the Polaris becomes a supernova or if it dies now, it will take 434 years in the future from now to know about it.

This is simply because ‘one light year is the distance traveled by light in one year’. Hence currently, one cannot know about what happened to the Polaris supernova even if it happens now.

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The perception of pain throughout the body arises when neural signals originating from the terminals of nociceptors are propagated to second-order neurons in the spinal cord or brainstem, whereupon they are transmitted to specific higher-order brain areas (Price, 2000). Recent studies have begun to elucidate some of the molecular mechanisms underlying the transduction of noxious stimuli. Many stimuli have been found to activate ion channels present on nociceptor terminals that act as molecular transducers to depolarize these neurons, thereby setting off nociceptive impulses along the pain pathways (Price, 2000; Costigan and Woolf, 2000). Among these ion channels are the members of the transient receptor potential (TRP) family. To date, the most studied member of the TRP family is the TRPV1 receptor. This is because it is the only one activated by capsaicin, the compound in chili pepper responsible for its “hot” taste; also, inhibiting TRPV1 has been shown to have therapeutic value (DiMarzo et al., 2002; Cortright and Szallasi, 2004). Although we will focus on the presence of these channels in nociceptors, we note that they have been identified in many other cell types and in various cortical and subcortical areas (Toth et al., 2005).  The transient receptor potential vanilloid 1 (TRPV1) channel is predicted to have six transmembrane domains and a short, pore-forming hydrophobic stretch between the fifth and sixth transmembrane domains (see Figure 5.1A). It is activated not only by the vanilloid capsaicin (Caterina et al., 1997), but also by noxious heat (>43°C) and low pH (Caterina et al., 1997; Tominaga et al., 1998), voltage (Gunthorpe et al., 2000; Piper et al., 1999), and various lipids (Julius and Basbaum, 2001; Caterina and Julius, 2001; Clapham, 2003; Cortright and Szallasi, 2004, Szallasi and Blumberg, 1999; Prescott and Julius, 2003; Jung et al., 2004; Bhave et al., 2003). In cells, TRPV1 is inactivated by its binding to PIP2 and is released from this block by PLC-mediated PIP2 hydrolysis (Prescott and Julius, 2003). TRPV1 is widely expressed in central nervous system (CNS) tissue and highly expressed in sensory neurons of the dorsal root ganglion [19]. This receptor also localizes to neurons that line the oral and nasal cavities [10], where it is found in a subpopulation of sensory afferent nociceptive nerve fibers [20].

(this is for question 1.)

Explanation:

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