The answer to the question above is a cell.
Sand is a granular material composed of finely divided rockand mineral particles. It is defined by size, being finer than gravel and coarser than silt. Sand can also refer to a textural class of soil or soil type; i.e., a soil containing more than 85 percent sand-sized particles by mass.[1]
The composition of sand varies, depending on the local rock sources and conditions, but the most common constituent of sand in inland continental settings and non-tropical coastalsettings is silica (silicon dioxide, or SiO2), usually in the form of quartz. The second most common type of sand is calcium carbonate, for example, aragonite, which has mostly been created, over the past half billion years, by various forms of life, like coral and shellfish. For example, it is the primary form of sand apparent in areas where reefs have dominated the ecosystem for millions of years like the Caribbean.
Sand is a non-renewable resource over human timescales, and sand suitable for making concrete is in high demand.[2] Desert sand, although plentiful, is not suitable for concrete, and 50 billion tons of beach sand and fossil sand is needed each year for construction.
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The insects clustered around the light is an example of A response to a stimulus. The insects are attracted to the light, so the flocked towards it.
Answer:
1. Inhibiting IP3 channels, leading to decreased Ca2 in the sarcoplasm and reduced contraction.
2. Increasing the relative activity of MLCP, leading to a decrease in tension.
3. Activating K channels, increasing K leaking out of the cell which hyperpolarizes it and decreases the likelihood of Ca2 entry.
Explanation
In smooth muscle, cyclic AMP (cAMP) mediates relaxation because cAMP inhibits a specific kinase required for myosin light chain protein (MLCP) phosphorylation, thereby triggering contraction in the smooth muscles. It has been shown that cAMP inhibits 1,4,5-trisphosphate (IP3)-dependent calcium ions (Ca 2+) release by activation of the cGMP-dependent protein kinase (PKG). PKG proteins act to modulate Ca2+ oscillations by stimulating sarcoplasmic Ca2+-ATPase membrane proteins, increasing Ca2+ in the sarcoplasmic reticulum stores and Ca2+ efflux from the cells, and activate voltage-gated potassium (K) channels, thereby leading to membrane hyperpolarization and reducing Ca2+ entry through Ca2+ channels.