Answer:
In allosteric inhibition, a regulatory molecule binds to a location other than the active site, resulting in a change in enzyme shape that allows the active site to bind substrate.
Explanation:
Allosteric regulation of an enzyme can be positive or negative, but it always involves effector molecules that bind to non-active site of the enzyme and change its conformation. That site of binding is called allosteric or regulatory site. If the enzyme activity is enhanced effector molecule is called allosteric activator but if the activity is decreased effector molecules are allosteric inhibitors.
It depends on which virus you are referring to specifically. Viruses come in many shapes and types; their variations are tremendous.
For HIV and Influenza, viruses acquire their envelops after maturation, during the budding off or detachment stage from the host cell.
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I think its Uracil replaces thymine on rna
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<span>The gradual thickening of the uterine lining also called Endometrial Hyperplasia is often caused by having excess estrogen without enough progesterone to balance it. During menstruation, the estrogen causes the lining to thicken in preparation for pregnancy. When the progesterone is made during the second half of the cycle and the progesterone levels drop, the lining is shed. And if the ovulation didn’t occur, progesterone is not made and the lining is not shed properly, the lining will keep on building gradually that may lead to cancer in some women. </span>
