Options for the question are not given. They are as follows :
A. Mate them and see what color the offspring are—that will be the dominant color
B. Mate them and see what color the offspring are—the other will be the dominant color
C. Mate them, and then mate their offspring to see what color the next generation is
D. Mate them together, and then mate their offspring to see what color the next generation is—the other color will be the dominant color
Answer:
A. Mate them and see what color the offspring are—that will be the dominant color
Explanation:
Out of the two colors, one will be recessive and one will be dominant. Recessive color will be homozygous since it is only expressed in homozygous condition. It is also given that in this cross the dominant color is also in homozygous condition.
When the guinea pigs mate, the resulting progeny will obtain one dominant allele from the dominant color parent and one recessive allele from the recessive color parent. The progeny will be heterozygous and show the dominant phenotype since the dominant allele will mask the recessive allele. Thus, the color which is visible in their offspring will be the dominant color.
Biomes are described in terms of abiotic factors like soil type and climate, and biotic factors factors like plant and animal life.
1. Latitude and the heat transported by winds are two factors that affect global climate.
2. Other factors, among them an areas proximity to an ocean or mountain range also influence climate.
Long-term potentiation (LTP) is considered a cellular correlate of learning and memory. The presence of G protein-activated inwardly rectifying K(+) (GIRK) channels near excitatory synapses on dendritic spines suggests their possible involvement in synaptic plasticity. However, whether activity-dependent regulation of channels affects excitatory synaptic plasticity is unknown. In a companion article we have reported activity-dependent regulation of GIRK channel density in cultured hippocampal neurons that requires activity oF receptors (NMDAR) and protein phosphatase-1 (PP1) and takes place within 15 min. In this study, we performed whole-cell recordings of cultured hippocampal neurons and found that NMDAR activation increases basal GIRK current and GIRK channel activation mediated by adenosine A(1) receptors, but not GABA(B) receptors. Given the similar involvement of NMDARs, adenosine receptors, and PP1 in depotentiation of LTP caused by low-frequency stimulation that immediately follows LTP-inducing high-frequency stimulation, we wondered whether NMDAR-induced increase in GIRK channel surface density and current may contribute to the molecular mechanisms underlying this specific depotentiation. Remarkably, GIRK2 null mutation or GIRK channel blockade abolishes depotentiation of LTP, demonstrating that GIRK channels are critical for depotentiation, one form of excitatory synaptic plasticity.
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<span>The correct answer is protein.</span>
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T2 virus is also known as Enterobacteria phage T2. This virus is a bacteriophage, which means that infects and kills E. coli. It consists of DNA and protein: the genome is linear double-stranded DNA, with repeats at either end and the protective coat is protein.</span>
