Answer:
cells lose their ability to maintain their sodium-potassium pumps, and eventually, they die.
Explanation:
If glycolysis is interrupted, these cells lose their ability to maintain their sodium-potassium pumps, and eventually, they die. The last step in glycolysis will not occur if pyruvate kinase, the enzyme that catalyzes the formation of pyruvate, is not available in sufficient quantities
High concentration and passive transport
Answer:
The correct answer is a acyl-carnitine readily cross the mitochondrial inner membrane but the acyl CoA do not.
Explanation:
Fatty acids are activated to form fatty acyl CoA by the help of ATP and CoA SH.But the so formed Acyl CoA cannot cross or pass the inner mitochondrial membrane to enter the mitochondrial matrix to initiate beta oxidation of fatty acid.
To overcome this problem fatty acyl CoA is converted to acyl carnitine and the so formed acyl carnitine readily crosses the inner mitochondrial membrane and enters the mitochondrial matrix.
Joint hypermobility syndrome (JHS) and Ehlers-Danlos syndrome, hypermobility type (EDS-HT) are the tissue disorders which can be characterized by chronic pain, joint instability complications, and minor skin changes. During these, Fatigue and headache are the very common symptoms; though they can be diagnosed using specific criteria.
JHS/EDS-HT is a rare common condition that could not be diagnosed by most clinicians and pain specialists, resulting in interventions like symptomatic and non-satisfactory treatments, because there is a lack of reasonable pathophysiologic rationale.
Pain, fatigue, and headache in JHS/EDS are usually treated with the help of certain symptoms or on the basis of doctors’ experience.
<span>Therefore, in order to the cure of such symptoms, doctors suggest pathogenic mechanisms. The major aim of the re-writing of the natural history of JHS/EDS-HT is to raise awareness among clinical geneticists and specialists treating chronic pain conditions about pain and other complications of JHS/EDS-HT.</span>
Answer:
D
Explanation:
when when lakes receive an overabundance of nutrients they can become polluted by excessive amount of algae.
