Answer: pathogen–host coevolution
Explanation:
A major driver of evolution is Reciprocal coevolution between host and pathogen. Rather than pathogen, one-sided adaptation to a nonchanging host, high virulence specifically favoured during pathogen–host coevolution. In all of the independent replicate populations under coevolution, the pathogen ( B. thuringiensis ) genotype BT-679 with known nematocidal toxin genes of C. elegans and high virulence specifically swept to fixation but only some of them go under one-sided adaptation,
so relative change in B. thuringiensis virulence was greater than the relative change in C. elegans resistance is due to the elevated copy numbers of the plasmid containing the nematocidal toxin genes
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In order to improve fat digestion, large fat globules must first be dispersed into smaller droplets in a process called <u>emulsification.</u>
<h3 /><h3>What is emulsification in the digestive system?</h3>
Fat emulsification is the process of increasing the surface area of fats in the small intestine by grouping them into small clusters. Large lipid globules are split up into a number of smaller lipid globules. In the chyme, these tiny globules are widely dispersed rather than aggregating into larger groups. Hydrophobic compounds include lipids. Bile salts, are present in bile and have both hydrophilic and hydrophobic sides.
Due to the fact that lipases can only effectively act on lipids when they are broken down into small aggregates, emulsification is crucial for the digestion of lipids. The lipids are converted into fatty acids and glycerides by lipases.
Learn more about emulsification here:
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Stable elements are those which have atleast one isotope with no tendency to "decay", i.e., change into anotherelement.
I think that would depend because there are 3c different forms of Trypanosoma brucei but for the most all three range between the size of 12-27<span>µm. </span>
