I think this should be done by yourself if it's your paragraph you can not be marked for plagiarism
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Since Br-80 does not exist, maybe that means Br-79 or Br-81 have very unequal abundances. For example, Br-79 may have 75% abundance whereas Br-81 may have 25% abundance.
CPT-I: Carnitine Translocase: CPT-II
- Carnitine palmitoyl transferase II deficiency is an autosomal recessively inherited genetic metabolic disorder characterized by an enzymatic defect that prevents long-chain fatty acids from being transported into the mitochondria for utilization as an energy source.
- The disorder presents in one of three clinical forms: lethal neonatal, severe infantile hepatocardiomuscular and myopathic.
What Is Carnitine Palmitoyltransferase II Deficiency?
Carnitine Palmitoyltransferase II (CPT II) deficiency, caused by mutations in the CPT2 gene, is an inherited disease in which the body cannot convert long-chain fatty acids into energy to fuel the body. There are three forms of the disease, and the severity and symptoms vary based on the form. In all three forms, symptoms can be triggered by periods without eating (fasting).
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When KOH and Cu(NO3)2 are mixed it yields copper(II) hydroxide and potassium nitrate. The balanced reaction is:
Cu(No3)2(aq) + 2 KOH(aq)<span> = Cu(OH)2(s) + 2 KNo3(aq)
</span>
As we can see from the equation a solid is formed therefore a
precipitate is formed which is the copper (II) hydroxide which has a blue to
purple appearance. This can be observed since copper
(II) hydroxide has a low
solubility in aqueous solution.
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According to the diagram and the arrows, a drop from level 4 (n = 4) to level 2 (n = 2) produces orange visible light.