Answer: Extracellular [Ca2+]
Explanation:
The sensitivity and density of the alpha receptors serve to <em>enhance the response to the release of</em> <em>norepinephrine (NE)</em> . However, they do not exert a strong influence as the concentration of calcium ions on the amount of <em>norepinephrine (NE)</em> released by sympathic nerve terminals.
The release of neurotransmitters depends more on either an external or internal stimulus.This results in an action potential which on reaching a nerve terminal, results in the opening of Ca²⁺ channels in the neuronal membrane. Because the extracellular concentration of Ca²⁺ is greater than the intracellular Ca²⁺ concentration, Ca²⁺ flows into the nerve terminal. This triggers a series of events that cause the vesicles containing <em>norepinephrine (NE)</em> to fuse with the plasma membrane and release <em>norepinephrine (NE)</em> into the synapse. The higher the action potential, the higher the Ca²⁺ flow into the terminals resulting in higher amount of <em>norepinephrine (NE)</em> into the synapse, and vice versa.
Catechol-O-methyltransferase (COMT) is one of several enzymes that degrade catecholamines such as dopamine, epinephrine, and norepinephrine. It serves a regulatory purpose to lower the concentration of norepinephrine upon its release from nerve terminals.
The reactivity depends on the elements valence electrons. The need to be stable will make an element become more reactive, In order for an atom to be stable, it needs 8 valence electrons in its outer shell.
Since Calcium is in group 2, it has 2 valence electrons, and would rather give up the electrons. While Sulfur has 6, it would need to gain 2 more to be stable, making it more reactive.
Much of biological studies are actually chemistry because of chemical changes that occur within organisms. Similarly, large parts of chemistry are governed by physics.
Ok hi what is the question