The given complex ion is as follow,
[Ru (CN) (CO)₄]⁻
Where;
[ ] = Coordination Sphere
Ru = Central Metal Atom = <span>Ruthenium
CN = Cyanide Ligand
CO = Carbonyl Ligand
The charge on Ru is calculated as follow,
Ru + (CN) + (CO)</span>₄ = -1
Where;
-1 = overall charge on sphere
0 = Charge on neutral CO
-1 = Charge on CN
So, Putting values,
Ru + (-1) + (0)₄ = -1
Ru - 1 + 0 = -1
Ru - 1 = -1
Ru = -1 + 1
Ru = 0
Result:
<span>Oxidation state of the metal species in each complex [Ru(CN)(CO)</span>₄]⁻ is zero.
0.116 V is the e value for the oxidation of cytochrome c by the cue redox center in complex iv when the ratio of cyst c (fe3 ) /cyst c (fe2 ) is 20 and the ratio of cue (cu2 )/cue (cu ) is 3.
<h3>
Explain the process of oxidation of cytochrome c.</h3>
When cytochrome c is oxidized by mitochondrial cytochrome oxidase (COX), it attaches to Apaf-1 to produce the apoptozole, which activates pro-caspase-9 and causes cell death. Cyst can be created from cytosolic cytochrome c. In the IMS, oxidized cytochrome c can scavenge superoxide without converting it into H2O2, a process that happens naturally but is accelerated by SOD. The benefit of scavenging superoxide independently of H2O2 synthesis is reducing the possibility of hydroxyl radical generation via the Fenton reaction.
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