Answer:
The answer is reciprocal chromosomal translocation
Explanation:
The Philadelphia chromosome (Ph) is the truncated chromosome 22 generated by the reciprocal translocation t(9;22)(q34;q11) and was first identified in 1960 in a patient with CML [3]. Translocation of the proto-oncogene tyrosine-protein kinase (ABL1) gene located on chromosome 9 to the breakpoint cluster region (BCR) gene located on chromosome 22 results in a BCR-ABL1 fusion gene on the Ph [4, 5]. Three BCR-ABL1 fusion gene hybrids encode BCR-ABL1 protein isoforms p210, p190, and p230, which have persistently enhanced tyrosine kinase (TK) activity. These aberrantly activated kinases disturb downstream signaling pathways, causing enhanced proliferation, differentiation arrest, and resistance to cell death [6, 7]. Tyrosine kinase inhibitors (TKIs) targeting the BCR-ABL1 protein are the most successful targeted therapy for Ph-positive leukemia.
That is called Urogenital myiasis which is a parasitic infestation. These flies are in the Diptera species group and the cause of this myiasis is from the eggs and larvae of this species (fly).
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Answer:
Parasympathetic nervous system effects are slow or inhibit function, where is sympathetic, is responsible for the intense physical activity.
Explanation:
The autonomic nervous system or ANS is divided into 2 distinct systems on the basis of their effects- the parasympathetic nervous system and the sympathetic nervous system.
Both of these have opposite actions on the functions that they maintain.
The sympathetic nervous system regulates the body for a high level of physical activity and also known as fight or flight. It fasts the functions of the body according to the situation.
The parasympathetic nervous system is having the opposite effect of the sympathetic nervous system slows high energy functions and relaxes the body.
Thus, the comparison of these nervous systems is mention above.