Answer:
The correct answer will be:
1. Acetylcholine binds to receptors on the motor end plate
2. Chemically-regulated ion channels open, causing depolarization
3 End plate potentials trigger action potential(s).
4. Transverse tubules convey potentials into the interior of the cell
5. Ca++ is released from the sarcoplasmic reticulum
6. Ca++ binds to troponin, pulling on tropomyosin
7. Binding sites on actin are uncovered, allowing myosin to bind and carry out power strokes
8. Force increases.
9. Ca++ is pumped (re-sequestered) into the sarcoplasmic reticulum
10. Force decreases.
Explanation:
The muscle contraction is a highly controlled mechanism which begins at the neuromuscular junction with the release of the acetylcholine neurotransmitter. This neurotransmitter causes the depolarization of the membrane by binding to the receptors of the motor end plate which generates an action potential. This action potential is transmitted via T-tubules from sarcolemma to the sarcoplasmic reticulum.
The sarcoplasmic reticulum releases calcium ions which binds to the troponin protein. This troponin removes the protein tropomyosin from the actin causing the rotation of the tropomyosin exposing the binding sites for myosin. The myosin binds to the actin using energy from the ATP which pulls the actin causing contraction. Another ATP binds the myosin head which weakens the bond between myosin and actin which releases the myosin which decreases the force between them decreases and the muscles relax.
D, calcium helps the bones
This scenario describes planter fascial strain. It can be managed through several precautions.
- Our body's strongest ligament is the plantar fascia. This is a band of connective tissue that runs from the base of the heel bone to the front of the foot and is made up of hundreds of collagen fibers.
- When we place our foot on the ground, this ligament, which is an elastic band, is stretched out and serves as a shock absorber.
- When we push our foot against the ground to quicken our pace, play, or leap, we might use the stored energy in the stretched plantar fascia.
- Localized soreness and pain over the plantar fascia, which runs from the heel to the forefoot under the foot, are signs of a plantar fascia strain.
- At the time of the injury, there may be a sudden, acute pain under the heel or arch of the foot. It's possible to feel a bump or nodule along the plantar fascia, which could be a sign of a partial rupture.
- Rest, ice, compression, elevation, wearing supportive shoes and orthotics constantly are all part of the treatment plan.
- After a full rupture, surgical tear reduction may be necessary, especially if a quick return to sport is crucial. In order to give the injury time to heal, rest is crucial.
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Answer:
it very important to lean about the anatomy and physiology so you can be ready for any situation.
Explanation:
Hypocalcemia is a curable illness that occurs when the calcium levels in your blood are too low. By including calcium in your diet on a daily basis, you may avoid calcium deficiency disease.
Hypocalcemia can be caused by a variety of medical disorders, but it is frequently caused by low amounts of parathyroid hormone (PTH) or vitamin D in your body. Hypocalcemia can induce numbness and tingling (particularly around the lips, hands, and feet), as well as muscular cramps. Your blood calcium level will be evaluated at your first post-operative appointment, and you may be weaned off the supplementary calcium prescription following your surgery. Keep in mind that calcium rich meals, such as dairy products, might include a lot of saturated and trans fat.
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