Answer:
S phase
Explanation:
The S is short for synthesis, which means that DNA is being replicated. This is one of the stages of interphase, when the cell is growing and getting ready to divide. Well, this is the getting ready to divide part of interphase; the cell needs to duplicate its DNA so it has enough to divide out to daughter cells.
When the swimmer is unable to take in enough oxygen for the sprint, the ATP (Adenosine triphosphate) will be compromised. In such conditions, the body will use anaerobic respiration to produce the ATP. Anaerobic respiration takes place in the absence of oxygen. It used the electron acceptors in place of the oxygen in the respiratory cycle. The amount of the ATP produced is, however, less in anaerobic respiration.
Hence, the answer is 'Option C - Anaerobic respiration would be used to produce small amounts of ATP in the absence of oxygen'.
Answer:
PFFT this might help? sorry if not mate
Explanation:
Cell cycle checkpoint controls play a major role in preventing the development of cancer [see Sherr, 1994, for a more detailed discussion]. Major checkpoints occur at the G1 to S phase transition and at the G2 to M phase transitions. Cancer is a genetic disease that arises from defects in growth-promoting oncogenes and growth-suppressing tumor suppressor genes. The p53 tumor suppressor protein plays a role in both the G1/S phase and G2/M phase checkpoints. The mechanism for this activity at the G1/S phase checkpoint is well understood, but its mechanism of action at the G2/M phase checkpoint remains to be elucidated. The p53 protein is thought to prevent chromosomal replication specifically during the cell cycle if DNA damage is present. In addition, p53 can induce a type of programmed cell death, or apoptosis, under certain circumstances. The general goal of p53 appears to be the prevention of cell propagation if mutations are present. The p53 protein acts as a transcription factor by binding to certain specific genes and regulating their expression. One of these, WAF1 or Cip1, is activated by p53 and is an essential downstream mediator of p53-dependent G1/S phase checkpoint control. The function of p53 can be suppressed by another gene, MDM2, which is overexpressed in certain tumorigenic mouse cells and binds to p53 protein, thus inhibiting its transcriptional activation function. Other cellular proteins have been found to bind to p53, but the significance of the associations is not completely understood in all cases. The large number of human cancers in which the p53 gene is altered makes this gene a good candidate for cancer screening approaches.
Answer:
A
Explanation:
BECAUSE LOOK AT THE PEDIGREE. IT SHOWS U
