Explanation:
Mutations increase variation within a population.
During the process of cell division, spontaneous changes within the genome can arise. These mutations are errors occur when copies of the DNA within the cell are made; mutations may range from small changes called single nucleotide polymorphisms, to large scale deletions, and additions which span multiple genes. There are two types:
- somatic: these only occur within certain cells, and arise from environmental factors such as UV light
- hereditary: occur within germ cells of the parent and later the fertilized egg which forms a zygote; these are present within all cells of the new organism.
Further Explanation:
During reproduction other events, such as crossing over during mitosis and meiosis, mutations lead to increases in genetic variation. This variation refers to the genetic characteristics present within a species. Mutations may be either beneficial or deleterious; they are maintained within cells, as they form new traits called alleles. Beneficial mutations may confer traits that increase the fitness of a species, along with ensuring survival by conferring a protective advantage- these phenotypic differences between organisms are called adaptations.
Sequences of DNA make up genes which can have different forms called alleles. DNA, which makes up the genotype, is transcribed into mRNA and later translated into amino acids which are linked together by rRNA to form proteins which make up the phenotype of an organism.Mutations in DNA sequences affect the corresponding mRNA and thus the protein encoded.
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The answer is B. CO2 + H2O + Sunlight ----> Glucose + O2
Answer:
- Calcium binds to troponin C
- Troponin T moves tropomyosin and unblocks the binding sites
- Myosin heads join to the actin forming cross-bridges
- ATP turns into ADP and inorganic phosphate and releases energy
- The energy is used to impulse myofilaments slide producing a power stroke
- ADP is released and a new ATP joins the myosin heads and breaks the bindings to the actin filament
- ATP splits into ADP and phosphate, and the energy produced is accumulated in the myosin heads, starting a new cycle
- Z-bands are pulled toward each other, shortening the sarcomere and the I-band, producing muscle fiber contraction.
Explanation:
In rest, the tropomyosin inhibits the attraction strengths between myosin and actin filaments. Contraction initiates when an action potential depolarizes the inner portion of the muscle fiber. Calcium channels activate in the T tubules membrane, releasing <u>calcium into the sarcolemma.</u> At this point, tropomyosin is obstructing binding sites for myosin on the thin filament. When calcium binds to troponin C, troponin T alters the tropomyosin position by moving it and unblocking the binding sites. Myosin heads join to the uncovered actin-binding points forming cross-bridges, and while doing so, ATP turns into ADP and inorganic phosphate, which is released. Myofilaments slide impulsed by chemical energy collected in myosin heads, producing a power stroke. The power stroke initiates when the myosin cross-bridge binds to actin. As they slide, ADP molecules are released. A new ATP links to myosin heads and breaks the bindings to the actin filament. Then ATP splits into ADP and phosphate, and the energy produced is accumulated in the myosin heads, which starts a new binding cycle to actin. Finally, Z-bands are pulled toward each other, shortening the sarcomere and the I-band, producing muscle fiber contraction.
Answer:
A) 50%
Explanation:
Boxes 2 and 3 are heterozygous because solving the punnet square results in those boxes being Tt
