Answer:
The kinetic theory of matter explains that all matter is made of particles which are constantly moving/have energy. The energy varies depending on the temperature of the matter, therefore determining if the matter is solid, liquid, or gas. When the energy of a particle is increased or decreased, a phase change can occur.
The bond angle varies depending on the type of bond between carbon atoms. For a single bond, its type is the sp³ hybridization. For this type, the bond angle between the carbon atoms is 109.5°. For sp² hybridization (double bonds), the bond angle is 120°. For sp hybridization (triple bonds), the bond angle is 180°. Thus,
A. This is an alcohol which consists of single bonds. So, the angle is 109.5°.
B. The angle is 180° because of the triple bond.
C. H₂C=C=CH₂ consist of a double bond, so the angle is 120°
Explanation:
Let us assume that the ratio for the given reaction is 1:1.
Therefore, we will calculate the moles of as follows.
Moles of solution = molarity × volume (L)
= 0.0440 M × 0.014 L
= 0.000616 moles
Moles of excess EDTA = 0.000616 moles
Also, the initial moles of EDTA will be calculated as follows.
Total initial moles of EDTA = 0.0600 M × 0.025 L
= 0.0015
Therefore, moles of EDTA reacted with will be as follows.
= 0.0015 - 0.000616
= 0.00088 moles
Since, we have supposed a 1 : 1 ratio between and EDTA
.
So, moles of = 0.00088 moles
Now, we will calculate the molarity of as follows.
Molarity of solution =
=
= 0.015 M
Thus, we can conclude that the original concentration of the solution is 0.015 M.
- A car travels 120km due to North .
- At last the car reached on town B from town A .
- The car's total displacement from town A to B .
✍️ See the attachment diagram .
✯ Hence, Total displacement from town A to town B is “ <u>8</u><u>2</u><u>k</u><u>m</u> ” .
Answer:
Some parameters in the population genetics of mutations*.
Naturally, a review of this length cannot cover all aspects of the population genetics of mutations. For example, mutation plays a pivotal part in coalescent theory (Hein et al. 2005) and in the construction of genotype–phenotype maps that are at the core of some efforts to understand adaptive landscapes, which provide a paradigm for understanding many broader aspects of population genetics from the perspective of individual mutations (‘causes cancer or not’), as reviewed elsewhere (Loewe 2009). Here we focus almost entirely on how populations of individuals are changed by large numbers of mutations that have specified effects on fitness.
In §2 of this paper, we discuss what is known about the diversity of mutations, and here and subsequently we refer to other papers in this themed issue that provide more in-depth information. In §3, we review some of the relevant theory in population genetics, starting with (i) simple theories that treat the fate of individual mutations in isolation before turning to more complicated models that consider (ii) linkage, (iii) epistasis, (iv) quantitative genetics approaches, and (v) challenges faced when attempting to integrate all these. Subsequently, we provide an overview of several general questions that have been resolved and others that remain (§4) and finally some conclusions (§5).2