Answer:
The answer to your question is 0.10 M
Explanation:
Data
Molarity = ?
mass of Sucrose = 125 g
volume = 3.5 l
Formula
Molarity = moles / volume
Process
1.- Calculate the molar mass of sucrose
C₁₂H₂₂O₁₁ = (12 x 12) + (1 x 22) + (16 x 11)
= 144 + 22 + 176
= 342 g
2.- Convert the mass of sucrose to moles
342 g of sucrose ------------------- 1 mol
125 g of sucrose -------------------- x
x = (125 x 1) / 342
x = 0.365 moles
3.- Calculate the molarity
Molarity = 0.365 / 3.5
4.- Result
Molarity = 0.10
The circulatory system moves blood in our bodies. The blood can move oxygen and nutrients to our muscles and limbs. The heartbeat pushes the blood, and the blood cells carry glucose.
Intermolecular forces in solids are strongest than in liquids and gases. Gases have the least strong intermolecular forces. Intermolecular forces are weak and are significant over short distances between molecules (determined by Coulomb’s law). The farther away from the molecules the weaker the intermolecular forces. Since molecules in solids are the closest, the intermolecular force between them as the strongest. Conversely, since gas molecules are farthest apart, the intermolecular forces between them are the weakest.
A ground state electron configuration follows the Aufbau Principle that states that electrons should be filled up in orbitals in increasing energy. In the given sequences, the right configuration is
<span>1s2 2s2 2p6 3s2 3p6 4s2 3d8.
2) the possible confirmation that follows Aufbau's principle is
D. </span><span>-[Kr] 5s24d105p3</span>
Cyclic nucleotides cAMP and cGMP are part of almost all major cellular signaling pathways. Phosphodiesterases (PDEs) are enzymes that regulate the intracellular levels of cAMP and cGMP. Protein kinase A or cAMP-dependent protein kinase mediates most cAMP effects in the cell. Over the last 25 years, various components of this group of molecules have been involved in human diseases, both genetic and acquired. Lately, the PDEs attract more attention. The pharmacological exploitation of the PDE’s ability to regulate cGMP and cAMP, and through them, a variety of signaling pathways, has led to a number of new drugs for diverse applications from the treatment of erectile dysfunction to heart failure, asthma, and chronic obstructive pulmonary disease. We present the abstracts (available online) and selected articles from the proceedings of a meeting that took place at the National Institutes of Health (NIH), Bethesda, MD, June 8–10, 2011.
